These deregulated B and T cells have the ability to make anti antibodies against red blood cells called autoimmune hemolytic anemia, against white blood cells called systemic lupus, against platelets needed for proper blood clotting called autoimmune thrombocytopenia plus many more.
How and why does this happen? This protective immune system turning against the body has been referred to as Plechner’s syndrome. What is this syndrome?
This syndrome occurs with the development of a cortisol deficiency or the production of defective cortisol. This leads to an elevated estrogen that not only binds the receptor sites of thyroid hormone but further binds any active cortisol and deregulates the B and T cell so that they can now function to the detriment of the patient.
In all actuality, the only true deficiency that does occur is in the B cells production of antibody. When this occurs and the level of mucous membrane antibody drops below 65 mg %, the patient has problems absorbing oral medication. This antibody is identified as IgA. Medications often fail due to this inability of the oral medication to reach the blood stream.
With any autoimmune disorder, intramuscular or IV injections, along with the use of thyroid hormones, will be more effective and maybe the only answer. Once the IgA levels have returned to normal, so proper absorbing of oral medications can take place, the use of oral medications can be used.